Objectives: To determine the effect of smoke on the dentinoenamel complex (DEC). Methods: the longitudinal slices of pulpodentinoenamel complex of the same human tooth realized by the aid of a 0.1 mm carborandum disc mounted on a micromotor (speed 30.000 turns per minute) are pierced at the apices with a 1 mm Ø diamond bur. This apical aperture serves as a stenopeical passage for the 0.2 mm nylon thread. These slices are stored in a 10-volume hydrogen peroxide for 24 hours, then agitated in distilled water for 10 seconds, stored in physiological solution for one hour and then conserved in alcohol at 95%. The experimentation is then realized using 20 ml plastic tubes (Lab*902/02/…University of Bordeaux 2). The slices are divided into two groups of experimental ( DEC smoked for 45 mn like a smoked food) and control (non-smoked DEC). The citric acid anhydryde is diluted in distilled water in a manner to obtain a concentration of 23% and share out to have 10ml in all test tubes. The slices are then introduced in test tubes for experimentation: Group 1 (control) : 23% citric acid + DEC Group 2 (experimental) : 23% citric acid + DEC smoked for 45 mn. Results: the effect of smoke on the DEC is significant within the two groups (P<.000 Friedman ANOVA). At the fourth hour the first signs of changes (enamel acid attack) occurred on the tooth slice of the test tube 2 (smoked DEC): mean = 257,9mn ± 24,36 S.D. The tooth slices of control group (non-smoked DEC) started to show changes at the ninth hour: mean= 540,5 mn ± 1,027 S.D. Conclusion: the smoke accelerates the citric acid destructive effect on the dentinoenamel complex.

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